Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Alternatively, our ability to rescue SERT function in KI midbrain synaptoneurosomes via the inhibition of FAK suggests elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by greater pFAK localization in 5-HT synapses. Our https://shermanz087dpa8.wizzardsblog.com/profile